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There is evidence of increased risk of persistent pulmonary hypertension of the newborn (PPHN), neonatal adaptation syndrome (jitteriness, respiratory distress, feeding trouble, persistent crying), and rare reports of congenital heart defectsthough absolute risk is still low

For example, the HPH model only reflects the pathological characteristics of hypoxia-induced PH, and cannot simulate the core disease characteristics of PAH such as the genetic background of IPAH (e.g., BMPR2 mutation) and the hemodynamic abnormalities of CHD-PAH, making it difficult to fully reveal the regulatory rules of UPS in PAH