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Regular glutathione and SOD are degraded and lost through digestion, making oral delivery ineffective

Pharmacological or genetic glutaminase regulation along with oxidative chemotherapy can help to improve the design of combination strategies that escalate the rate of therapeutic success in cancer patients

To date, no small molecule exists that is demonstrated to inhibit the TRF1:TIN2 interaction

Animal-model studies have explored its effects on ischemia-reperfusion injury, fistula healing, and angiogenic vessel formation in chronic wound preparations

However, the pros and cons of the treatment do leave us perplexed and hesitant