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Recent technological developments allow for the interrogation of CpG methylation status across the genome, and this has uncovered associated CpG sites within candidate genes including those that have not been previously implicated in cigarette smoking ( AHRR , CYP1A1 ), are differentially methylated, suggesting that smoking effects might be targeted to specific regions of the epigenome

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These dynamics suggest that interventions aimed solely at increasing awareness of health risks may have limited impact unless they also address the underlying attitudes and social influences that sustain vaping behavior (34, 35)

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