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However, during other metabolic activities, NAD+ is consumed by NAD+-dependent enzymes and may subsequently become depleted over time, contributing to increased DNA damage, age-related illnesses and diseases, and mitochondrial malfunction

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Certain laboratory reports suggest that Pal-GHK could reduce processes related to protein glycation caused by reactive species such as acrolein or malondialdehyde, thus potentially contributing to structural preservation in extracellular proteins

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Icariin treatment significantly inhibited the activity of apoptotic factors caspase-3 and Bcl-2-associated X protein (Bax)/Bcl-2, and reduced intracellular ROS and NO levels [67]