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The inflammatory cytokines released by an excessive inflammatory response increase glial activation (Watkins et al., 2014

Oxidative stress and antioxidant defense in migraine A study using a migraine rat model revealed increased malondialdehyde levels in the cortex and trigeminal ganglion, along with pro-injurious effects of reactive oxygen species (ROS) on the brain via transient receptor potential A1 (16)

At day 3 after ICH, IF upregulated the expressions of Nrf2 and HO-1 in microglia which were attenuated by Sirt3 deletion (Fig

Conversely, a growing body of evidence supports the involvement of ferroptosis in renal IRI [29, 30]

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