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Neuroscience 145 (1), 256264

Additionally, this study underscores the need for clinical approaches that address both the toxicological aspects and modulation of the inflammatory response to improve patient outcomes

A.KuhnJ.LevineJ

These proinflammatory molecules also induce the above lesions through the nuclear factor-B (NF-B), Janus kinase/signal transducers and activators of transcription (JAK/STAT), and TGF-/Smad signaling pathways[99-101]

The benefits appeared to be slightly stronger in healthy individuals (rather than those with medical problems), when single organisms were used (rather than combinations), and when the treatment duration was eight weeks or longer