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Therefore, UBB+1 is involved in both secretase-dependent and independent pathways for A generation and this could be a reasonable explanation as to why UBB+ conjugated protein aggregation has been found to be associated with A plaques in transgenic UBB+1 AD brain (van Leeuwen et al., 2006

Interestingly, recent studies have proposed that DNA methylation can lead to the exclusion of a histone variant (H2A.Z), preferentially located at active gene promoter 36,37

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